Regulation of fetal gene expression in heart failure - ScienceDirect
Cardiac foetal reprogramming: a tool to exploit novel treatment targets for the failing heart - Pol - 2020 - Journal of Internal Medicine - Wiley Online Library
Comparative Analysis of mRNA Isoform Expression in Cardiac Hypertrophy and Development Reveals Multiple Post-Transcriptional Regulatory Modules | PLOS ONE
AFos Dissociates Cardiac Myocyte Hypertrophy and Expression of the Pathological Gene Program | Circulation
MicroRNAs in the Human Heart | Circulation
Fixing nature's genetic mistakes in the womb | Clinical & Translational Science Institute
JCI - Toward transcriptional therapies for the failing heart: chemical screens to modulate genes
Multifaceted roles of miR-1s in repressing the fetal gene program in the heart | Cell Research
Harnessing fetal and adult genetic reprograming for therapy of heart disease. | Semantic Scholar
Defining the Fetal Gene Program at Single-Cell Resolution in Pediatric Dilated Cardiomyopathy | Circulation
Reactivation of the fetal gene program. Quantitative RNA dot blot... | Download Scientific Diagram
Regulation of fetal gene program by the calcineurin-NFAT-mediated... | Download Scientific Diagram
Frontiers | Transcription Factors Involved in the Development and Prognosis of Cardiac Remodeling
Fetal gene therapy for neurodegenerative disease of infants | Nature Medicine
Roles of Cardiac Transcription Factors in Cardiac Hypertrophy | Circulation Research
Proposed RVF model consisting of reactivation of a ROR2 fetal gene... | Download Scientific Diagram
A Systematic Review of Fetal Genes as Biomarkers of Cardiac Hypertrophy in Rodent Models of Diabetes | PLOS ONE
Cardiac foetal reprogramming: a tool to exploit novel treatment targets for the failing heart - Pol - 2020 - Journal of Internal Medicine - Wiley Online Library
Frontiers | Why and How Imprinted Genes Drive Fetal Programming
JCI - The H3K9 dimethyltransferases EHMT1/2 protect against pathological cardiac hypertrophy
Cardiac foetal reprogramming: a tool to exploit novel treatment targets for the failing heart - Pol - 2020 - Journal of Internal Medicine - Wiley Online Library
A human fetal liver-derived infant MLL-AF4 acute lymphoblastic leukemia model reveals a distinct fetal gene expression program | Nature Communications
A) Expression of the fetal gene program (ANF, BNP, bMyHC, SERCA) in... | Download Scientific Diagram
Changes in cardiac hypertrophy are associated with fetal gene program... | Download Scientific Diagram
